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Since vinculin plays a key role in regulating gut motility, damage to it can cause a permanently weakened MMC, reducing the guts ability to clear bacteria and archaea and setting the stage for chronic, relapsing SIBO

accumulating evidence suggests that it promotes tumor-associated macrophage polarization toward an M2-like immunosuppressive phenotype, thereby reinforcing a tumor-permissive microenvironment [23, 24]

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The catalytic tetrad formed by Sec, glutamine, tryptophan and asparagine is essential for GPx activity, since these residues are crucial for enzyme-substrate interaction and stabilization of the GSH-GPx interaction [47, 65, 66]

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