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However, inadequate NADPH production in cells with restricted glucose intake significantly augments actin cytoskeletal disulfide bonds, causing disruption in the actin network and intracellular disulfide hyperplasia

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10.1079/bjn20061729 Summary Keywords biliary atresia, hepatotoxicity, hypermethioninemia, methionine metabolism, metabolic intermediates Citation Jiachen Z, Paul Kwong Hang T, Kenneth Kak Yuen W and Vincent Chi Hang L (2023) Pathological role of methionine in the initiation and progression of biliary atresia