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Type 1 is an autoimmune disease in which insulin-producing -cells are attacked by the hosts immune system, and is thought to be caused by a combination of viral infection, an environmental trigger, or gene malfunction [3, 4]

In the absence of GPX4 being available to counter this process and remove lipid hydroperoxides, a runaway feedback cycle could arise that will ultimately destabilize the membrane, leading to pores and micellization

Subcutaneous injection of each of the four peptides significantly improved skin phenotype in terms of the clinical skin scores for erythema and scale compared to the IMQ group

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As shown here, other substrates like SDF-1 and BNP should also be taken into account and may help to better understand the therapeutic potential of DPP4 inhibitors