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Although interventions aimed at enhancing mitochondrial biogenesis, restoring mitochondrial and FAO homeostasis, and employing remote ischemic preconditioning have demonstrated potential in mitigating AKI progression, further investigation is required to address unresolved concerns related to their safety and clinical efficacy

RIP1/RIP3-regulated necroptosis as a target for multifaceted disease therapy (Review)

Jain and Parsanathan, 2020)

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