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The literature reports that carnitine can promote the excretion of excess organic or fatty acids in patients with defects in fatty acid metabolism and/or specific organic acidopathies that bioaccumulate acylCoA esters.1-6 Secondary carnitine deficiency can be a consequence of inborn errors of metabolism or iatrogenic factors such as hemodialysis

Nutrition as medicine with regard to mitochondrial health is evidenced by numerous studies related to the beneficial effects of phytochemical-rich food and fermented food items