demonstrated that AS-IV reduces ROS accumulation in ISO-induced cardiomyocytes, mitigates mitochondrial membrane damage, counters Ca 2+ redistribution due to oxidative stress, inhibits calpain-1 activation, maintains calcium homeostasis, and prevents apoptosis in ISO-induced myocardial hypertrophy, reducing arrhythmia risk (Mei et al., 2015)
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This approach demonstrated high feasibility for early in vivo diagnosis of ACI/AKI, detecting these conditions at least 24 and 48 hours earlier than standard clinical assays for ACI and AKI, respectively
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