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On the basis of the crystal structures of Ib CO 22 and Vv CO 23 , it was initially believed that this residue is responsible for the differentiation between mono- and diphenolase activity by restricting access to the active center 31

10.1073/pnas.0911187107 Proc

on the other hand, it inhibits ferritinophagy by disrupting the interaction between NCOA4 and FTH1, reducing Fe 2+ release and mitochondrial ROS generation and ultimately exerting critical liver protective effects (Wang J