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Gropman AL, Summar M, Leonard JV (2007) Neurological implications of urea cycle disorders

Delivery of ketone ester in a preclinical model of SARS-CoV-2 infection in K18-hACE2 mice 45 reduced the dependence on glucose but enhanced the potential of CD4 + T cells to oxidize amino acids and fatty acids, increasing their production of IFN and viral clearance (Fig

There are several concerns in this field: firstly, despite the association of the mitochondria-derived peptide MOTS-c with bone metabolism and current studies confirming the effects of MOTS-c intervention, the role and mechanism of endogenous skeletal MOTS-c in regulating bone metabolism have not been evaluated

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