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NLY01 also significantly diminished pro-inflammatory cytokine (TNF-, IL-1, IL-1, and C1q) expression in the hippocampus, preventing the stimulation of reactive astrocytes

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Given that KRAS mutations are prevalent in almost 30% of non-small cell lung cancer (NSCLC) patients [2], we chose a Kras G12D -driven spontaneous lung cancer murine model to elucidate the detailed mechanism by which DDX3X regulates lung cancer progression, which might be also extended to KRAS wildtype lung cancers