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These studies illustrate broad investigative interest, but they also underscore that effects vary by model and mechanism

Several drugs have been studied, but none have demonstrated significant benefits, indicating a need for larger, well-designed, controlled trials

In wound infections, TLR signaling is activated by PAMPs such as lipopolysaccharides (TLR4 ligands), lipoteichoic acid (TLR2 ligands), flagellin (TLR5 ligand), and unmethylated CpG DNA (TLR9 ligand) derived from invading bacteria, fungi, or viruses [32]

These ROS molecules, which include superoxide and hydrogen peroxide, cause oxidative damage to cells by destroying lipids, proteins, and DNA (51)

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