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This mechanism means many patients on GLP-1 therapy already achieve caloric deficits and metabolic benefits similar to fasting, even during eating windows

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Also, more generally, development of non-glutamate drugs for pain and depression has been plagued by failures in advanced human trials

That matters when the research question concerns GLP-1-specific signalling, gastric emptying kinetics, -cell insulin secretion, or hypothalamic POMC activation, without GIP or glucagon receptor confounders

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