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This suggests that direct COP1-p53 recognition may require additional cofactors, adaptors, or specific cellular signal conditions such as glucose induce assembly of CRL4 COP1 for p53 degradation, as suggested in Su et al 29

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Tang and Dai (2014a, 2014b), showed that the glutamate application on brain slices intensified biophotonic activities and this activity could be blocked by oxygen and glucose deprivation

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