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The compound reduces pro-inflammatory cytokines (TNF-alpha and IL-6), limiting excessive tissue damage that prolongs recovery

Conversely, pathological factors such as protein aggregation and proteopathic stress, and possibly the influence of systemic comorbidities, accelerate brain ageing, leading to a decline in the neuronal reserve over time [132,139]

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Prespecified and Integrated Subgroup Analyses from the RECONNECT Phase 3 Studies of Bremelanotide