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As two negative regulators of p53, MDM2 and MDMX facilitate ferroptosis with or without p53, most likely by altering the lipid profile of cells (Venkatesh et al., 2020), which is confirmed by evidence that with the treatment of MDM2 inhibitor nutlin-3, p53 expression increases and leads to the suppression of X c system inhibitor-induced ferroptosis in HT-1080 cells (Tarangelo et al., 2018)

Bloodwork can even confirm this, showing low albumin and other markers consistent with a starvation state

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Furthermore, exenatide has received orphan drug designation for the treatment of IIH from the European Medicines Agency and the US Food and Drug Administration