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Mitochondrial dysfunction and damage can ultimately lead to impaired neuronal function, abnormal synaptic transmission, impaired synthesis and release of neurotransmitters, and even neuronal death, resulting in cognitive dysfunction

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Sources conflict on the exact relationship: some describe fosgonimeton as a phosphate prodrug of Dihexa, while Athira has described a structurally distinct molecule so it is most accurate to call it the same program, structurally distinct, with conflicting public descriptions

A clustering of contamination events or persistent post-inflammatory hyperpigmentation cases tied to specific at-home protocols would shift the recommendation toward more conservative cadence or stricter contraindication screening

In animal studies, inhibiting NNMT has been associated with altered energy handling in fat cells and with preserved SAM and NAD availability