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In related research, murine models with elevated lipid serum profiles showcased an increased secretion of IL-6 from CD1b+ dendritic cells, suggesting its role in modulating T cell orientation and heightening IL-17A output by HJ1 T cells (64)

M., & Zanivan, S

Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired