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Illustratively, BPC 157 induced the release of NO on its own, which was quite resistant to the NOS blocker L-NAME, counteracted the adverse effect of both the NO blockade and NO over-stimulation in many models and species (for a review, see [13]), and interacted with NO-specific molecular pathways [54,58] (i.e., BPC 157 regulates vasomotor tone and the activation of the Src-Caveolin-1-endothelial nitric oxide synthase pathway [54])