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The LKKTET(Q) motif binds G-actin, buffering monomers and regulating polymerization so cells can rapidly reorganize their cytoskeleton and move into injured tissue
Peptides are fragile
Clinically, BPC-157 and TB-500 are synergistic
It is also frequently studied for its protective effects on the gastrointestinal tract, particularly in models of gastric ulceration, inflammatory bowel disease, and NSAID-induced damage
Upon reception of a signal to migrate such as chemical mediators called chemokines present near the edge of the wound the bound pool of actin monomers becomes immediately available to form actin filaments in the lamellipodium and filopodia of migrating cells
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