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These agents potentially have complementary effects in cardiovascular and renal protection (Graphical Abstract)
Nishiyamas mechanistic signals revealed incretin-mediated suppression of progesterone production, a pathway with considerable ramifications for luteal function and endometrial receptivity, highlighting the significance of this discovery [31]
in EBioMedicine builds upon previous research on the FOXO4-p53 complex, a critical regulator of SCAPs
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