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The astrocytes modify their phenotype, adopting a cellular morphology characterized by hypertrophic nuclei and cell bodies with distinct long and thick processes with increased glial fibrillary acidic protein (GFAP)

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Summarily, we suggest that retatrutide enlarged FOC in HAP via the cAMP system through its cognate receptors

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Iron uptake and the labile iron pool (LIP) Extracellular Fe3+ typically binds to transferrin and enters cells via endocytosis through transferrin receptor 1 on the cell membrane

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