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These compounds exert neuroprotective effects by mitigating oxidative stress, suppressing neuroinflammation, inhibiting tau hyperphosphorylation, and reducing amyloid-beta aggregation

[5] [4] The mechanism of action involves binding to GLP-1 receptors found throughout the body, particularly in the pancreas, gastrointestinal tract, and brain

EPA and DHA also give rise to anti-inflammatory compounds that "turn off" the inflammatory response

(2013) Betaine supplementation decreases plasma homocysteine in healthy adult participants: a meta-analysis, Journal of chiropractic medicine, Vol

Adverse safety signals or unexpected long-term complications associated with chronic GLP-1 receptor agonist therapy could necessitate label revisions or therapeutic limitations that reduce addressable patient populations and clinical adoption rates