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In this study we assessed the in vitro ability of OligoG alone, and in combination with nystatin, to inhibit planktonic growth, perturb biofilm formation on glass and clinical grade silicone surfaces and disrupt established biofilms of a wide range of candidal clinical isolates currently on the WHO fungal priority pathogens list

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Each targeting distinct mechanisms of gut barrier repair, inflammatory modulation, and immune regulation

Compared to DB group, GLP-1RA group exhibited 1067 upregulated and 410 downregulated genes (Fig

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