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By the 2010s, athletes adopted it for its tissue-repairing effects, supported by preclinical data

The accumulation of APs and AGEs in combination with -amyloid glycation can lead to cellular dysfunction and the progression of neurodegenerative and metabolic diseases 58

There have been reported at least 15 syndromes in which carnitine deficiency seems to be secondary to genetic defects of intermediary metabolism or to other conditions [40]

Foods that digest slowlylike high-fiber vegetables, lean protein, and healthy fatssupport the medication by extending gastric emptying and controlling appetite

I will be straightforward: I do not have a clear mechanistic explanation to offer here