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The results showed that the transient expression of SsCm1 inhibited callose deposition and the expression of pathogenesis-related genes ( NbPR1a and NbPR2 ) induced by PAMPs, including Flg22, chitin, and Sclerotinia culture filtrate elicitor (SCFE), while no significant variation was detected in the ROS burst induced by PAMPs (Supplementary Fig

My triggers were tiredness, combined with not drinking/eating enough or getting stressed

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(8) reported significantly higher mean and adjusted IMA levels in AA patients than in the control group, indicating a potential association between IMA and the imbalance of oxidative stress

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