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This destructive oxidative process is considered a central mechanism of ferroptosis, iron overload accelerates the production of ROS via the Fenton reaction, which in turn amplifies lipid peroxidation, compromising the cell membrane and leading to ferroptosis

These findings together imply that upregulation of Prxs renders motor neurons less susceptible to neurodegeneration, whereas breakdown of this redox system at late disease stages induces neuronal degeneration and accelerates disease progression (Kato et al., 2005)

Adding GHK-Cu introduces powerful matrix synthesis and gene modulation effects

Li Y, Wang RY, Deng YJ, Wu SH, Sun X, Mu H

In parallel, the higher but transient P53 expression in Gstp1/2 / mice could contribute to the maintenance of the redox homeostasis and cell survival as well as the early increase in GSTM1 protein levels that blocks the TRAF2/ASK1 signaling pathway