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showed that ARID1A-deficient cancer cells expressed low levels of the key cystine transporter SLC7A11 and thus exhibited low basal levels of GSH, which made these cancer cells specifically vulnerable to inhibition of the GSH metabolic pathway 25

Raducka-Jaszul O, Bogusawska DM, Jdruchniewicz N, Sikorski AF

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5B), suggesting that DNA methylation played a role in the mechanism of DMF regulation of CBS gene expression