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Oral administration of NAC was previously reported to improve photoreceptor function in rd10 mice [12] and in Phase 1 clinical trial [13], outlining a new promising minimally invasive route for RP treatment

The -synuclein aggregates specifically bind to the translocase of outer mitochondrial membrane (TOMM) 20, significantly inhibiting the transmembrane transport of mitochondrial precursor proteins, ultimately leading to mitochondrial dysfunction in dopaminergic neurons [96]

They carry potential side effects - commonly including nausea, vomiting, diarrhea, constipation, and abdominal pain - and require medical supervision

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