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The entire injected/systemic evidence base is preclinical rodent in-vivo studies and in-vitro cell and biochemical experiments

Based on these findings, we propose that Erastin-induced cell death in EMS tissue is not solely limited to ferroptosis but is also accompanied by necroptosis, further elucidating the complex interplay between these cell death pathways in EMS pathophysiology

Expression of glycosaminoglycans and small proteoglycans in wounds: modulation by the tripeptide-copper complex glycyl-L-histidyl-L-lysine-Cu

This metabolic byproduct also downregulates antigen-presenting capacity, ultimately compromising the ability of DCs to initiate effective T-cell responses (139)

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