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Table 2 summarises the design, demographic, intervention methods, and reproductive endpoints of the research included in this review

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Experimental model for production of perforating duodenal ulcers by cysteamine in the rat

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This pathway activation enables: Enhanced neuronal survival under stress conditions Increased dendritic arborization and synaptic density Improved axonal sprouting and regeneration Activation of neuronal repair mechanisms Studies using neuronal cell cultures revealed that Cerebrolysin significantly increases neurite outgrowth and protects against excitotoxic injury, effects attributed to neurotrophic factor-like activities[2]

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