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Experimental evidence in rats and mice highlights the neuroprotective potential of GLP-1 medicines, primarily through anti-inflammatory and cytoprotective mechanisms 13 and via metabolic improvements in the CNS (Figure 1)

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Furthermore, ROS interact with PUFAs present in cellular lipid membranes, resulting in producing numerous lipid peroxides that impair the integrity of cell membranes and ultimately result in cell death (17)

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In more severe cases, oral medications may be necessary