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Similarly, when phosphatidylcholine (PC) synthesis is compromised by myeloid-specific deletion of CTP:phosphocholine cytidylyltransferase alpha (CCT), the rate-limiting enzyme in PC synthesis, macrophages fail to secrete the pro-inflammatory cytokines TNF- and IL-6 in response to LPS, most likely due to secretory defects of the ER and Golgi ( Figure 3 )