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Growing interest in GLP-1 diabetic neuropathy reflects a broader shift toward understanding microvascular complications beyond heart disease

doi: 10.3390/molecules27072244 60 FengSTangJWeiXLuZXuYZhangTet al

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Another plausible explanation was that CD4+ T cells with high D3R expression were specific for -synuclein, thus these cells could only be detected at the site of inflammatory infiltration, instead of the peripheral circulation (124)

HMG-CoA reductase inhibitor attenuates experimental autoimmune myocarditis through inhibition of T cell activation