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However, resistance to enzalutamide has now also been identified as a result of an Phe876Leu point mutation in the LBD (213) and the expression of NH 2 -terminal domain splice variants (163) in CRPC, emphasizing the need for continued research and development of strategies to switch off androgen receptor signaling

These markers cover the treatable drivers of diffuse shedding, and they are most useful read together rather than one at a time

Last reviewed 2026-07-25

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