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To examine GLP-1 dependence, we used the gld-2 gld-1 null double mutant meiotic entry defective background ( Introduction ) and compared foci distribution relative to wild type in gld-2 gld-1 with either glp-1(+) or the glp-1 null allele q175

In vivo rodent models have explored tissue-specific IGF-1R engagement by comparing the biodistribution and signalling footprint of IGF-1 LR3 against the native 70-amino acid ligand

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