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Both modifications sit in the region that IGF binding proteins recognise, and the functional consequence is a large loss of IGFBP affinity while type 1 IGF receptor binding is largely preserved

SalB effectively prevents oxygen radical generation and cardiomyocyte apoptosis via the TRIM8/recombinant glutathione peroxidase 1 (GPX1) signaling pathway both in vivo and in vitro ( SalB -mediated myocardial protection

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