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Although the mechanism explaining why C-KIT is pathogenetically important in sun-protected melanomas remains poorly understood, it does not prevent the protein from being a potentially effective therapeutic target and a series of C-KIT inhibitors are currently under pre-clinical and clinical investigations (82, 8789)

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In the current study, the PTZ group indicated an upregulation of proBDNF levels but a significant decrease in mBDNF expression in relation to its precursor proBDNF when compared to the control group

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