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In macrophages exposed to OxLDL, GHRP-2 is hypothesized to reduce lipid accumulation, further illustrating its potential antioxidative and protective roles against proatherogenic agents

Both interventions target aspects of metabolic health, which has led to interest in whether their combination might enhance outcomes beyond what either achieves alone

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The interplay between CoQ redox balance, AMPK activation and ETC directionality suggests that the ferroptotic outcome of complex I inhibition depends not only on its biochemical activity but also on the metabolic state of the cell