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We hypothesized that the GLP-1R agonist, liraglutide, might alleviate chondrocyte apoptosis and ECM degradation by regulating ER stress

Therefore, collectively, these findings suggest that administering NAD + precursors prevents oxidative stress and mitochondrial dysfunction, and hence may be a useful therapeutic target for ALS

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Treatment with semaglutide resulted in amelioration of neuronal loss, with significantly higher numbers of neurons observed in the treated group compared to untreated controls in the cortex (S1BF)