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La sintesi endogena avviene a partire dal substrato TML (6-N-trimetillisina), che a sua volta deriva dalla metilazione dell'amminoacido lisina

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ONOO can cause nitration and oxidation of tyrosine residues in proteins, leading to altered protein function and signaling [32]

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KYN levels were elevated (~ 2.1-fold), and TDO expression (both glial density and intensity in grey and white matter) was significantly higher, suggesting that enhanced KP metabolism may contribute to psychotic features in bipolar disorder