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The TRAP telomerase assay confirms that TERT activation is the upstream driver: TERT knockdown (siRNA) abolishes Epitalons anti-senescence effect, while TERT overexpression (viral vector) phenocopies it without Epitalon establishing TERT as the essential effector

When combined with BPC-157, TB-500 enhances recovery at both the cellular and structural levels, creating a synergistic healing effect

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C.BlecharzK.MagriniS.WagnerJ.Nieminen-KelhM.et al

Associated with this mechanism, we can also mention the negative influence on the recapture of excess Glu by glutamate transporter 1, but also the increase in the activity of phospho-extracellular signal-regulated kinases (pERK 1/2), which has an immediate effect, including the marked increase in the extracellular level of Glu, activation of microglia, and initiation of neuroinflammatory processes [86,87,88]