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Beyond the p53 KO-mediated adipocyte-autonomous shift towards a catabolic gene programme, ATMs can also affect adipocyte lipid release by inducing catecholamine resistance 3 , which is defined as a reduced catabolic response to physiological levels of catecholamines and can be caused by lowered expression of -adrenergic receptors 79

Glioma-associated microglia and macrophages/monocytes display distinct electrophysiological properties and do not communicate via gap junctions

Most researchers discard cloudy peptide solutions because aggregation reduces cellular uptake by 4060%, even when the peptide remains chemically intact

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[DOI] [PMC free article] [PubMed] [Google Scholar] 75.Grunnet M, Sorensen JB