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To determine whether inflammation-mediated NAD + depletion had any effect on mitochondrial function in this model, an XFe96 Seahorse assay was conducted to measure mitochondrial oxygen consumption rates (OCRs) (46)

Thus, general LPO, which develops in all cell membranes, cannot always be considered as a marker of ferroptosis

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This can be attributed to the replenishment of key vitamins and minerals that play a crucial role in brain function, such as B vitamins and magnesium

Glutaredoxin 2 catalyzes the reversible oxidation and glutathionylation of mitochondrial membrane thiol proteins: implications for mitochondrial redox regulation and antioxidant DEFENSE