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Physical activity prompts the movement of the fatty acid transporter (FAT/CD36) from its internal storage sites to the mitochondrial membrane in muscle cells (110, 111), where it engages with acyl-CoA synthetases that regulate the presence of fatty acyl-CoA for CPT1 (112), This indicates that FAT/CD36 could play a role in affecting how fatty acids are oxidized in mitochondria when engaging in physical exercise

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Targeting programmed cell death in metabolic dysfunction-associated fatty liver disease (MAFLD): a promising new therapy

doi: 10.3390/epidemiologia5030032 3

doi: 10.1038/s41419-019-1549-6 71 TangZLiCKangBGaoGLiCZhangZ