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Concomitant use of dapagliflozin resulted in less glycemic variability, prolonged TIR and improved patient satisfaction regarding with hyperglycemia

Metabolic analysis revealed that the source of the anti-ferroptosis effect of GCH1-overexpressing cells was BH4/BH2, supplementation with which rescued cell viability

This, in addition, requires the availability of mitochondrial NADPH, which also provides reducing equivalents for TrxR2 and is consequently vital for the functioning of the thioredoxin and peroxyredoxin systems

Subcutaneous injection self-administration [Online]