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Beyond the phagocytic respiratory burst, G6PD acts more generally as a key regulator of cellular redox homeostasis and provides substrate for NADPH oxidase by generating NADPH, thus exerting an innate immune modulatory role in these capacities

This work implies that maternal energy status may influence placental function in terms of steroidogenesis, given that the primary source of glucose for the placenta is derived from the maternal circulation

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