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The AKT pathway can phosphorylate more than fifty substrate proteins and modulate processes disrupted in PD, such as the enhancement of synapse formation, autophagy, and LTP, as well as inhibiting the secretion of pro-inflammatory cytokines, apoptosis, microglial activation, tau phosphorylation and the accumulation of -synuclein and A (Fig

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Protein-protein interactions, for example, often involve large surface areas that are challenging for small molecules to disrupt but may be addressable with appropriately designed peptides

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