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A higher dysregulation burden of brain DNA methylation in female patients implicated in the sex bias of schizophrenia
The GCH1BH4 system The GCH1BH4 pathway has been identified as an alternative ferroptosis defense mechanism independent of GPX4 and CoQH [26, 27]
Animal studies assessing exposure to high stress or trauma have also reported astrocyte dysfunction, which can lead to extracellular glutamate levels increasing to toxic amounts (i.e., excitotoxicity) (46)
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