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Further, inflammation in T2DM contributes to pancreatic carcinogenesis by promoting oxidative stress, activating the oncogenic pathways of NF-B and STAT3, and can drive tumor promoting processes such as apoptosis inhibition, cell cycle progression, and epithelialmesenchymal transition [21]

Additionally, it protects normal tissues from the toxic effects of the drug

[13] Tan PK, et al

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