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Studies in chicken embryos suggest that at least part of the mechanism by which BPC-157 promotes vascular growth is through the stimulation of VEGFR2, a cell surface receptor active in the nitric oxide signaling pathway [4], [7], [8]

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The preclinical signal for BPC-157 is unusually consistent across independent research groups and across a wide range of injury models, pointing toward a compound with genuine biological activity rather than a laboratory artifact

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