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Mechanistically, ferroptosis in transplanted hearts triggers the initial release of DAMPs, which bind to endothelial Toll-like receptor 4 (TLR4) and stimulate type I interferon production via Trif-mediated signaling, promoting the recruitment of neutrophils to the injured heart [103]

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For true cellular protection, taking capsules or liposomal forms internally is significantly more effective

This key molecule protects cells and mitochondria from free radical damage